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Total body potassium depletion and renal tubular dysfunction following carbenoxolone therapy
  1. R. J. Dickinson,
  2. R. Swaminathan

    Abstract

    A patient with hypokalaemia-associated renal tubular damage following carbenoxolone therapy is described. The hypokalaemia was reflected by a low level for total body potassium (TBK) and investigations confirmed renal tubular dysfunction. Hypokalaemic nephropathy may be secondary to intracellular potassium depletion rather than the hypokalaemia itself.

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