SELF ASSESSMENT QUESTION
Cardiology
Spontaneous T wave inversion after ventricular pacing: What is the probable mechanism?
Department of Cardiology, School of Medicine, Trakya University, Edirne, Turkey
Correspondence to:
Correspondence to:
Dr O Erdogan
Arseven Sitesi Konutlari No:2, D-100 Karayolu Fifi Yani, Edirne, 22030 Turkey; okanerdogan@yahoo.com
Accepted 3 March 2006
Answers on p e21
| The first 150 words of the full text of this article appear below. |
An 85 year old man with a history of hypertension and no known cardiac disorder was admitted because of recent onset dizziness, effort intolerance, and dyspnoea. A 12 lead surface electrocardiogram (ECG) taken on admission showed 2:1 second degree atrioventricular block with a rate of 40 beats/min. QRS complexes were narrow and T waves were upright in all leads. C reactive protein and serum troponin 1 concentrations taken 12 hours apart were normal. Subsequently, a temporary transvenous pacemaker lead was placed in the right ventricular apex because of low blood pressure (90/60 mm Hg) and heart rate (40 beats/min). Capture threshold was within the acceptable range. The temporary pacemaker was programmed to VVI mode and a lower rate of 70 beats/min (fig 1
). After a few days of continuous pacing, it was turned off and the patient’s own rhythm resumed (fig 2).
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Figure 1 Precordial leads showing right ventricular | |||||||||
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Spontaneous T wave inversion after ventricular pacing: What is the probable mechanism?
Postgrad. Med. J. 2006 82: e21.[Extract] [Full Text] [PDF]
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