Review
Hypophosphataemia in anorexia nervosa
L HåglinDepartment
of Social Medicine, University Hospital, SE-901 85 Umeå, Sweden
Correspondence to: Lena Håglin lena.haglin.us@vll.se
Submitted 30 December
1999;
Accepted 22 August 2000
| The first 150 words of the full text of this article appear below. |
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Introduction |
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The prevalence, causes, and consequences of hypophosphataemia in the clinical treatment of various diseases are described in the literature, but are not so seriously regarded as a severe electrolytical disturbance.1-3 The clinical conditions when hypophosphataemia should be suspected are listed in fig 1. There is a triad of disturbances: hypokalaemia, hypomagnesaemia, and hypophosphataemia which often follows trauma, and glucose overload.4 In anorexia nervosa patients, hypokalaemia, hypochloraemia, and metabolic alkalosis are commonly seen.5-7
| Figure Removed (Available Only in the Full Text) |
A high prevalence of hypophosphataemia is seen among post-traumatic and/or critically ill patients undergoing intensive care.8 Infectious diseases are also associated with the risk of developing hypophosphataemia.9 Immunological disturbances can result from a deficiency of several nutrients, such as zinc.10 Hypophosphataemia was found in anorexia nervosa patients with respiratory distress and signs of pneumonia.11-13 Recently, it was reported that the main causes of death of patients with anorexia nervosa was electrolytic disturbance and infection.11 14
The incidence of
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